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On a mechanistic level, influenza virus infection interferes with the cellular metabolic pathways of glycolysis, glutaminolysis, pentose phosphate, and fatty acid synthesis to generate ATP (adenosine triphosphate) and structural materials of viral replication (9)

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This may be because the GEK group was able to not only slowly release the active substances of EW (tryptophan, l-arginine) but also release high amounts of GHK-Cu with vascular activity, which is consistent with the results of the precellular experiments