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In autoimmune hepatitis and T-cell-mediated hepatitis models, gut microbiota-derived ICA induces the expression of PI3K-interacting protein 1 by activating AhR in CD8 + T cells, thereby inhibiting the PI3K/Akt/mTOR pathway, limiting excessive activation of effector T cells, and attenuating liver injury
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doi: 10.1016/0300-483x(90)90035-f
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