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2) Larger longitudinal studies are needed to determine whether selectively targeting CD38’s enzymatic activity to limit ADO production can enhance immune responses and improve outcomes in MM, and whether the resulting ADO/INO imbalance represents a metabolic signature of resistance
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Why Researchers Study It Because insulin-like growth factor signaling plays a central role in cellular growth, protein synthesis, and tissue regeneration, researchers use IGF-1 LR3 as a tool to study these pathways under more sustained and observable conditions than native IGF-1 allows
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