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The anti-inflammatory actions of MUFAs are based on their ability to counteract the effects of long-chain saturated fatty acids on hepatocytes, which include reducing endoplasmic reticulum stress, restricting lipotoxicity induced by accumulation of saturated fatty acids, decreasing ROS production, and inhibiting nuclear factor-κB (NF-kB) transcription factors by binding peroxisome proliferator-activated receptor γ (PPARγ) and G-protein coupled surface receptor 120 (GPR120) (138)
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doi: 10.1016/j.atherosclerosis.2007.06.009 [DOI] [PubMed] [Google Scholar] 56.López-Méndez I, Méndez-Maldonado K, Manzo-Francisco LA, Juárez-Hernández E, Uribe M, Barbero-Becerra VJ
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