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They are derived from primitive myeloid progenitor cells and migrate into the CNS during embryogenesis, appearing before E8 in mice and 4.5–5 weeks in humans (Lichanska and Hume, 2000
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This seems to be achieved through mechanisms such as dysregulation of intracellular calcium signaling, depolarization of mitochondrial membrane potential independent of CypD, and production of ROS
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