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This effect is attributed to its ability to modulate Heat Shock Protein expression (HSPs), thus impeding the crucial binding between heat shock factor and elements (HSF-HSE) necessary for the stress-induced transcription of stress genes (85, 86)
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Mira Meeus, Jo Nijs, Linda Hermans, Dorien Goubert & Patrick Calders (2013) The role of mitochondrial dysfunctions due to oxidative and nitrosative stress in the chronic pain or chronic fatigue syndromes and fibromyalgia patients: peripheral and central mechanisms as therapeutic targets?, Expert Opinion on Therapeutic Targets, 17:9, 1081-1089, DOI: 10.1517/14728222.2013.818657 Myhill S, Booth NE, McLaren-Howard J
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