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Glial-targeted anti-inflammatory agents such as minocycline, TLR4 antagonists and CCR2/CCR5 inhibitors have been shown to reduce pro-inflammatory cytokine release, limit peripheral immune-cell recruitment and protect synapses in experimental models of chronic ethanol exposure [11,57,58]
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Dung tích:30 ml
Types of necroinflammation, the effect of cell death modalities on sterile inflammation
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