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H2 receptor antagonists also affect B12 absorption, but less than PPIs

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these events may deplete GSH, covalently modify proteins, perturb mitochondrial function, and trigger inflammatory signaling if they occur in healthy compartments ( Practically, this means that toxicity packages for MSN-phenolic systems should include matched non-malignant controls, hemocompatibility, macrophage and endothelial stress readouts, organ-level biodistribution, and time-resolved oxidative injury markers rather than relying only on acute viability assays

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