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demonstrated that the polarization of macrophages into M1 and M2 subsets in mouse heart transplant models was critically dependent on the tumour-necrosis factor receptor associated factor 6 (TRAF6) and the mammalian target of rapamycin (mTOR) proteins, even though the exact mechanism seemed to be rather complex [250]

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Furthermore, the relevance of NQOs is based on their capacity to mitigate the noxious effect of several toxins and prooxidants [155]