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The results of ELISA manifested that Erastin induced increased levels of IL-6 and TNF-α, and decreased IL-10, indicating an inflammatory imbalance (Fig
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On the other hand, pro-apoptotic proteins, including Bax, antagonize anti-apoptotic proteins or translocate to mitochondrial membranes to form membrane-integrated homo-oligomers that induce mitochondrial pore formation, leading to the loss of MMP and resulting in the cytosolic release of apoptotic factors [43,44]
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